The subject of alimentary toxemia was discussed in London
nearly 80 years ago, before the Royal 'Society of Medicine by fifty-seven of
the leading physicians of Great Britain. Among the speakers were eminent
surgeons, physicians and specialists in the various branches of medicine. The
following is a list of the various poisons noted by the several speakers:
indol, skatol, phenol, cresol, indican, sulphurretted hydrogen, ammonia,
histidine, indican, urrobilin, methyImercaptan, tetramerhylendiamin,
pentamethyiendiamine, putreacin, cadaverin, neurin, coolin, muscarine, butyric
acid, beraimidazzolethy-lamine, methylgandinine, ptomarropinene, botulin,
tyramine, agamatine, tryptophane, sepsin, Idolehtylaymine, and
sulpherroglobine. Of the 36 poisons mentioned above, several are highly active,
producing most profound effects, and in very small quantities. In cases of
alimentary toxemia, some one or several of these poisons is constantly bathing
the delicate body cells and setting up charges, which result in grave disease.
There are now available some tests to check if someone has one or several of
those; they are:
1. Putrechrome Reaction (PCR):
for the detection oftoxic phenol (ethereal) species, putrefactive ketones,
putrescine, cadaverine, and acetoacetic acid. The toxic
effects of putrefactive bacteria cannot be underestimated. Thioether, and
ethereal organic sulfide, is a decidedly toxic creature.
The ethereal sulphates are conjoined with aromatic substances
belonging to the oxy-group, most important of which are the salts of phenol,
indoxyl, and skatoxyl As a rule their amounts vary with degrees of intestinal
putrefaction and hence this serves not only as a diagnostic link but helps to
figure the index of intensity.
There are certain conditions in which a relative increase or
an absolute decrease of total sulphates occurs. These should be remembered as
sulphate excretion is increased in practically all febrile states, pneumonia,
acute myeitis~ leukemia, diabetes mellitus~ carcinoma, progressive muscular
atrophy, and some types of the most difficult eczemas. The conjugate sulphates
may be noticed in intestinal toxemia as markedly increased~ as well as
carcinoma (increased) volwlus (increased), chronic nephritis (increased)~ and
obstructive jaundice (increased).
2. Indole/Skatole: Products
of intestinal putrefaction Indole is formed in the degradation of tryptophan.
It occurs naturally in human feces and has an intense fecal smell. Tryptophan
is first converted to indole, then to indican by bacteria in the gut. Indican
is then excreted into the urine and from there into the diaper. Skatole is
formed in the intestine by the bacterial decomposition ofLtrytophan and found
in fuca1 matter, to which it imparts its characteristic odor. Skatole has been
shown to cause pulmonary edema in goats, sheep, rats, and some strains of mice.
It appears to selectively target Clara cells, which are the major site of
cytochrome P450 enzymes in the lungs. These enzymes convert skatole to a
reactive intermediate~ 3-methyleneidolerine, which damages cells by forming
protein adducts. In a 1994 report released by five top cigarette companies,
skatole was listed as one of the 599 additives to cigarettes as a flavoring
ingredient. Its name is derived from skato, the Greek word for dung. Sweet!
3. Indican/Skatole: indicative,
when in quantity, of protein putrefaction in the intestine (indicanuria).
4. Sulfafloc: For
the detection of ethereal sulfates representing the highly toxic species like
methylmercaptan and other thioethers due to h'beration of bacteria found in the
oral cavity and large bowel.
5. Microalbuminuria proteinuria:
a highly sensitive test for the detection of slight amounts of albumin, a known
harbinger for pre-renal failure, and nephron damage by diabetes mellitus and
hypertension and detection ofproteids because of Leaky Gut Syndrome.
6. Calcium Test: Useful
for detection of calcium dumping syndrome. Urine when containing high amounts
of sodium ions along with calcium indicates a negative calcium balance,
especially in BAD (basic American diet) dietary, interstitial acidosis, and
7. Carcinochrome Reaction
(CCR): for the detection of pep tides in urine known to be
associated with the pre-cancerous state. Imagine, a urine test that points very
early on to the cancer diathesis.
Cancer diagnosis today centers on radiology and tumor markers.
Tumor markers are substances that can be detected in higher-than-normal amounts
in the blood or body tissues of some patients with certain types of cancer. A
tumor marker may be made by a tumor itself or by the body in response to the
tumor. Such a substance serves to "mark" the tumor; it is a "tumor marker."
But, of course, however, by the time a doctor finds a positive
tumor marker, it is generally already too late and te opportunity for
prevention has long passed.
The carcinochrome reaction according to GUTSCHMIDT, among
others, is an early detection test for the prediction of cancer. It is a urine
test based on the difference between cancer and normal metabolism, the food
products are normally oxidized to carbon dioxide, ammonia, and water. In
abnormal metabolism, the intermediary products of digestion are not fully
utilized. Diabetics, as we know, cannot fully utilize glucose; therefore it
spills into the urine. Patients with jaundice will spill bile into the urine
due to inflammation of the liver. Females with an inflamed cervix will display
abnormal cells called dyspleasia (abnormal tissue development) as seen on the
Cells, in a precancerous stage, will start spilling odd
proteins into the blood which spill through the kidneys and appear in the
urine. This is an important and vital feature. These odd proteins are actually
peptides of small molecular size, small enough to pass through the kidneys into